FOSL1 and nasopharyngeal carcinoma: AR-induced LINC01503 functioned as an oncogenic lncRNA during NPC tumorigenesis and progression through recruiting SFPQ to the FOSL1 promoter and activating its transcription; interestingly, the entire pathway could be blocked by the AR ligand antagonist Enz or activated by the AR ligand agonist DHT (Fig. 8i).