To determine whether LINC01503/SFPQ promotes NPC progression by activating FOSL1, we introduced a FOSL1 overexpression or empty vector in HK1 and SUNE1 cells with stable knockdown of LINC01503 (sh1503) or its control (shCtrl), as monitored by quantitative RT-PCR and western blotting (Supplementary Fig. 3). This evidence concerns the gene FOSL1 and nasopharyngeal carcinoma.