VPS4A and infection: Several trafficome targets identified here as high-ranking hits besides those mentioned above were previously shown to be not only involved in infection biology in STM in general, but specifically in SCV and/or SIF biogenesis including: dynein–DYNC1H1 [78–80], filamin–FLNA [81], myosin II–MYH10 [82], VPS4A/B [49].