To evaluate the clinical significance of CAMK2G in MM pathogenesis, we examined two independent microarray datasets from GEO databases (GSE5900 [13, 14], GSE13591 [15]) and found that elevated CAMK2G mRNA was associated significantly with disease progression from normal or clinically insidious stages to malignant MM (Figure 1A, 1B). This evidence concerns the gene CAMK2G and Miyoshi myopathy.