Thus, defective gut defense in cathelicidin-null mice was, at least in part, attributed to a reduced CXCL1-stimulated influx of neutrophils during infectious colitis and, LPS+LL-37 can drive epithelial chemokine responses at concentrations of LL-37 that were below microbicidal or cytotoxic.69 The gene discussed is CAMP; the disease is infectious colitis.