It is associated with massive recruitment and activation of neutrophils resulting in excessive production of proinflammatory cytokines and chemokines including IL‐1β, IL‐6, IFN‐γ, IL‐8, CXCL10, and CCL2, eventually leading to the development of ALI/ARDS (Jiang et al., 2018, 2019). Here, IL1B is linked to acute respiratory distress syndrome.