It has been previously shown that STAT1 can be phosphorylated by EGFR, a key tyrosine kinase relevant to the majority of tumors.32,33 We examined the EGFR status in meningioma tissues and cells, detecting high levels of pEGFR in both tumor lysates and meningioma cells, when compared to NMT and HMC (Figure 5A). The gene discussed is STAT1; the disease is neoplasm.