NOS2 and nephrocalcinosis: As expected, immunofluorescence and IHC assays revealed a worsened kidney interstitial inflammatory response with increased infiltration of M1Mφ (iNOS) in the nephrocalcinosis mouse model after intraperitoneal administration of antagomiR-93 as compared with the results of SFN treatment (Figure 6E, Figure S6J, Figure S7).