The study revealed that maternal physiology and behavior impacted child anxiety and three possible pathways were defined: augmentation of child anxiety through increased maternal salivary IgA, which led to enhanced child IgA; reduced social repertoire of the child due to reduced maternal oxytocin—and, in turn, reduced child oxytocin; and a direct impact of increased maternal anxiety on child anxiety. This evidence concerns the gene CD79A and interstitial lung disease specific to childhood.