In keeping with this, colonic macrophages from ILC-depleted mice showed an increase in Col6a1, Col6a2, and Col6a3 transcripts (Figure 6B) that together form trimers that make up collagen type VI, previously shown to promote myofibroblast activation in the context of lung fibrosis (Ucero et al., 2019). The gene discussed is COL6A2; the disease is pulmonary fibrosis.