COL4A1 and gastric cancer: The molecular mechanisms revealed that miR-29s inhibited gene expression by binding to the 3′ UTR region of COL4A1 gene in GC cells (Figures 4 and 5), and the COL4A1 expression was negatively correlated with the levels of miR-29s in clinical tissues of GC, which strongly indicated that the regulatory network of miR-29b/c-COL4A1 may be involved in the different growth pattern of GC.