The importance of dosing Lp(a) in FH patients was corroborated by Langsted et al. [11], reporting that the HR for myocardial infarction (MI), which is 1 for the individuals who are unlikely to have FH and Lp(a) < 50 mg/dL, rises to 1.4 in individuals who are likely to have FH and Lp(a) > 50 mg/dL, then to 3.2 in those with possible, probable or definite FH, and Lp(a) < 50 mg/dL, and finally to 5.3 in those with possibe or definite FH and Lp(a) > 50 mg/dL. This evidence concerns the gene LPA and myocardial infarction.