In a zebrafish TB model, using the fish‐adapted pathogen and close genetic relative of Mtb, Mycobacterium marinum (Mm), Hif‐1α stabilisation increased myeloid production of Il‐1β, vital for decreasing bacterial burden via neutrophil nitric oxide (NO) production [48, 49], alongside increasing pro‐inflammatory macrophage Tnfa production [50]. The gene discussed is HIF1A; the disease is tuberculosis.