To disrupt caveolae-mediated endocytosis, we pre-incubated FG cells with different concentrations of the cholesterol-binding reagents nystatin and filipin III, the tyrosine kinase inhibitor genistein, and the PKC activation inhibitor phorbol 12-myristate 13-acetate (PMA) before LCDV infection. Here, PRRT2 is linked to infection.