In a similar mechanism to NEAT1 modulation of the E2F3 pathway, MALAT1 was shown to directly interact and “sponge” miR-206, leading to activation of the Protein kinase B (AKT)/mammalian target of rapamycin (mTOR) pathway to promote epithelial-to-mesenchymal transition (EMT) and migration of A549 and H1299 NSCLC cells [34]. This evidence concerns the gene NEAT1 and non-small cell lung carcinoma.