Using SNP rs17009129, we find a causal link between FABP2 abundance and CAD (p-value 1.1x10−4; FDR <0.05; βMR -0.11; seMR 0.028; βMR and seMR units: log(OR)/standard deviation of residualised protein concentration) without significant heterogeneity (p-value 0.24) which suggests shared causal genetic control. Here, FABP2 is linked to coronary artery disorder.