The analysis showed that regardless of whether the ovarian cancer patients were genetically mutated or not (BRCA 1/2 mutation or HRD mutation), the addition of PARP inhibitors could extend the prognosis of this population, and that those with BRCA 1/2 mutation or HRD mutation had significantly longer prognosis than no gene mutation. This evidence concerns the gene PARP1 and ovarian carcinoma.