VEGFA and myocardial infarction: In a rat MI model, the increased levels of VEGFA released from AC-VEGFA-hMSCs promoted angiogenesis, decreased infarct size, and improved myocardial function, while the decreased levels of VEGFA secreted by nonAC-shVEGFA-hMSCs reduced angiogenesis, increased infarct size, and impaired cardiac remodeling.