MiR-181b is increased in human atherosclerotic plaques and abdominal aortic aneurysms (AAA), and inhibition of miR-181b suppressed the development and progression of atherosclerosis and aneurysms through increasing expression of TIMP3, elastin, and collagen (Di Gregoli et al., 2017). Here, ELN is linked to abdominal aortic aneurysm.