TIMP3 and myocardial infarction: MiR-17-92 cluster is able to induce cardiomyocyte proliferation in postnatal and adult heart as well as in response to myocardial infarction (Chen et al., 2013), which is partly caused by binding of miR-17 to 3’ UTR of Timp3 and suppressing Timp3 expression (Shi et al., 2017; Liu et al., 2018).