A recent study comparing transcriptome data of disease stabilization and tumor progression from a neuroblastoma patient after oncolytic virotherapy found that CXCL9/10-CXCR3 levels were significantly upregulated during tumor progression, underlining the dichotomous role of this axis, which was also shown to promote metastasis when active as an autocrine feedback-loop [40,41]. This evidence concerns the gene CXCR3 and neoplasm.