KRAS and neoplasm: In accordance with this, the functional examination of the adenosine diphosphate (ADP)-activated respiration rate (Vmax) and the mitochondrial outer membrane (MOM) permeability in patient-derived KRAS-/BRAF-mutated as well as wild-type tumor specimen indicates that KRAS mutations might be linked to an oxidative phenotype, while BRAF mutations to a glycolytic phenotype [38].