Individuals with mutations in IL‐17A, IL‐17 receptor A (IL‐17RA), or IL‐17RC (Puel et al, 2011; Ling et al, 2015; Levy et al, 2016) are susceptible to chronic mucocutaneous candidiasis (CMC) as are patients with dominant‐negative mutations in the transcription factor signal transducer and activator of T cells (STAT) 3 (Milner et al, 2008) and gain‐of‐function (GOF) mutations in STAT1 (Toubiana et al, 2016), which result in defects of Th17 cell differentiation. This evidence concerns the gene IL17RA and Chronic mucocutaneous candidosis.