STIM1 and experimental autoimmune encephalomyelitis: Mice with T cell‐specific deletion of ORAI1, STIM1, or both STIM1 and STIM2 had profound defects in Th17 cell function resulting in partial or complete protection from experimental autoimmune encephalomyelitis (EAE), a Th17 cell‐dependent murine model of multiple sclerosis (MS; Ma et al, 2010; Schuhmann et al, 2010; Kaufmann et al, 2016).