AKT1 and Miyoshi myopathy: The possible reason might include that miR‐125a was a negative regulator of PI3K/Akt/mTOR and NF‐kB signaling pathways, which lncRNA NEAT1 had stimulating effect on; therefore, miR‐125a was negatively associated with MM risk, ISS stage, biochemical indexes (β2‐MG, LDH), and prognosis in MM patients.31, 32