Molecularly, increased expression of genes involved in the MHC-I mediated antigen presentation (e.g., B2M, HLA-A, B, C, E), interferon signaling pathways (e.g., IFITs, IFI44/IFI44L, IFNARs, IFNGRs, CXCL9-11, STAT1), and apoptosis program (e.g., CASP8, TRAIL) were evidently detected in the OC3 tumor parts. The gene discussed is TNFSF10; the disease is neoplasm.