Interestingly, we have previously failed to find a relationship between LBP and LPS in 10 healthy overweight and obese males in the fasting state (r = −0.341, p = 0.369; unpublished data) and Ghamim et al. who used both LAL and LBP assays to measure ME failed to detect a fasting relationship between the two measures of endotoxemia [21]. The gene discussed is LBP; the disease is serum lipopolysaccharide activity.