TGFB1 and pulmonary fibrosis: From the first histopathological descriptions, the molecular basis of the pulmonary fibrosis progression due to SARS-CoV-2 infection is still unclear, and could be complex and multifactorial, involving direct viral effects, immune dysregulation/cytokines (MCP-1; IL-6, IL-8, TGF-β, TNF-α), and increased oxidative stress (Liu J. et al., 2020; Xu et al., 2020).