The most common genetic cause of this syndrome is gain‐of‐function mutations in a gene encoding a potassium channel (KCNT1), but recently, in a cohort of 135 patients with EIMFS, two were found with de novo heterozygous pathogenic variants in PIGA, an X‐linked gene involved in GPI‐anchor biosynthesis, implicating this pathway in this particular epileptic syndrome.21 The gene discussed is KCNA3; the disease is epilepsy of infancy with migrating focal seizures.