Given the importance of the innate immune system in controlling viral replication during the early stages of viral infection, we examined the levels of eosinophils (Siglec‐F+CD11c‐), neutrophils (Ly6G+CD11b+), DCs (I‐Ab+CD11c+), NK cells (CD3‐NK1.1+) and macrophages (CD64+) at the site of infection (BAL) and spleen at d5 and d7 pi. Here, FCGR1A is linked to viral infectious disease.