Most of the known BD are associated with oligogenic variations in the coding regions or the splice sites.3–8 However, both we and Ingo Kurth et al. (Franke et al., 2016) discovered that the duplications in 17q24.3 influencing the coding sequences of KCNJ2 and KCNJ16 and the noncoding elements 5′ of SOX9 could cause solitary brachydactyly‐anonychia. Here, KCNJ2 is linked to Behcet disease.