Since DDR1 phosphorylation could be responsible of growth inhibition (Saby et al., 2018), we evaluated the impact of nilotinib (50 nM), a receptor tyrosine kinase inhibitor with high potency against DDR1, on colon carcinoma cell proliferation using both control (Figure 4C) and HT-29DDR1–GFP (Figure 4D) cells. This evidence concerns the gene NTRK1 and colon carcinoma.