This hypothesis is further supported by the significant lower levels of lipocalin-2, a marker of intestinal inflammation linked to epithelial damage and neutrophil infiltration, recorded at 72 h post-infection in the stools of CAA1- and CCG4-treated animals in comparison to the control groups (infected/non-treated and infected/HGN194-treated groups) (Figure 3B). The gene discussed is LCN2; the disease is infection.