TNNI3 and Sepsis: The experimentalresults showed that myocardial dysfunction occurred in the rat model of sepsis.However, when miR-328 antagomir was injected, myocardial dysfunction in sepsisrats was reversed, LVSP and +dp/dtmax were significantly increased,levels of -dp/dtmax, LVEDP, cTnI, and CK-MB were significantlydecreased (P<0.001, Figure 3C-G).Therefore, it was determined by the rat sepsis model that miR-328 may be apotential mechanism for the regulation of myocardial function in sepsis.