Based on the differential expression of immune-related genes and apparent evasion mechanisms, they found that 77.4% of TNBC tumor specimens escape through transforming growth factor-beta (TGF-β), 48.0% through cytotoxic T-lymphocyte-associated protein 4 (CTLA4), 57.7% through decoy receptor 3 (DcR3), and 34.3% through programmed cell death-1 (PD-1). The gene discussed is TNFRSF6B; the disease is neoplasm.