SOD1 and schizophrenia: Considering the data presented above in the context of pathogenesis of schizophrenia, it seems that examining the effects of GSH deficiency in the early postnatal life, both on the activity of antioxidant enzymes (SOD, CAT, GPx, GR) and on ROS concentrations, may be of vital importance for defining the role of oxidative stress in the development of this disease.