Therefore, we examined the distribution and abundance of the MΦ2a (CD80−/CD163+/CD206+) and MΦ2b (CD80−/CD163+/CD86+) subtypes in controls and endometriosis in the CD14+low/CD68+low and CD14+high/CD68+high subpopulations (Supplementary Fig. 2a). This evidence concerns the gene MRC1 and endometriosis.