DPYSL2 and neuropathic pain: Therefore, here we aimed to study the populations of DRGs that express both CRMP2 and phosphorylated CRMP2 under naïve conditions as well conditions of a neuropathic pain model; the SNI model was chosen because we previously reported in this model of chronic neuropathic pain that CRMP2 phosphorylation by Cdk5 was increased in DRGs and in spinal cord [8].