Interestingly, a key characteristic of the mesenchymal subgroup of GBM is its strong association with immune‐related genes and an enrichment of infiltrating immune cells (Chen & Hambardzumyan, 2018; Behnan et al, 2019), thereby raising the possibility that mesenchymal‐specific features of the hGIC phenotype might be responsible for inducing mTOR signalling in TAM‐MG. Here, MTOR is linked to myasthenia gravis.