Interestingly, a key characteristic of the mesenchymal subgroup of GBM is its strong association with immune‐related genes and an enrichment of infiltrating immune cells (Chen & Hambardzumyan, 2018; Behnan et al, 2019), thereby raising the possibility that mesenchymal‐specific features of the hGIC phenotype might be responsible for inducing mTOR signalling in TAM‐MG. Here, MTOR is linked to glioblastoma.