IL10 and infection: Taken together, these data suggest that during a subpatent P. falciparum blood‐stage infection, monocytes responded to TLR stimulation with increased inflammatory cytokine production, while they co‐produced regulatory cytokines in response to restimulation with P. falciparum parasites, that is increased IL‐10 producers, suggesting that during subpatent P. falciparum malaria monocytes develop a regulatory capacity, potentially to minimise tissue damage following chronic activation.25