The MPTP-intoxicated group showed a considerable increment in the Bax/Bcl-2 ratio (p < 0.001) as compared to the control group whereas in CGA-treated PD mice, the Bax/Bcl-2 ratio (p < 0.001) was observed to be significantly reduced, which shows the antiapoptotic property of CGA in the parkinsonian mouse model (Figure 4(b)). This evidence concerns the gene BCL2 and Parkinson disease.