KIF2C and neoplasm: Our results run in parallel with theirs regarding PRC1, MAD2L1, ASPM, RACGAP1, CCNA2, MKI67, KIF2C, TPX2, BIRC5, TTK, NEK2 and CENPF, where they were all found to be overexpressed in CS-related tumours (Supplementary Table 6).