TNFSF12 and atherosclerosis: The binding of TWEAK to its initial receptor, Fn14 (fibroblast growth factor inducible molecule 14), which is mostly expressed in injured tissue, triggers an inflammatory response (e.g., by inducing interleukin 6 and 8 (IL-6/8) [3]) which has been shown to be involved in the formation and progress of several chronic inflammatory diseases, such as atherosclerosis [4].