In an examination of seven tumour types, CSTF2 was found to be up-regulated in five (lung adenocarcinoma, uterine corpus endometrioid carcinoma, bladder urothelial carcinoma, lung adenocarcinoma, and breast invasive carcinoma) and to exhibit a significant correlation between expression and 3′UTR shortening [141]. This evidence concerns the gene CSTF2 and lung adenocarcinoma.