This is unlikely, however, given the phenocopied tumor suppression phenotype, high degree of homology between MEIS1 and MEIS2 (72.12% similarity), and previously published data demonstrating that knockdown of both MEIS1 and MEIS2 is required for increased PrCa cell line aggression in vivo (Bhanvadia et al., 2018). The gene discussed is MEIS2; the disease is neoplasm.