The suggested common pathway among these three kinases (Wen et al., 2003; Schweitzer et al., 2012), along with the effect of AKT1, SGK1, and PP2B on HTT (Bowles and Jones, 2014; Saudou and Humbert, 2016), suggest that the one investigated in the present study may be a candidate pathway on the pathogenetic crosslink between DM and HD. This evidence concerns the gene SGK1 and Huntington disease.