Similarly, EBV infection appears to trigger expression of the HERVs that have been associated with MS pathogenesis, and for both the HERVs and herpesviruses significant cross reactivity between the viral protein epitopes, MOG (myelin oligodendrocyte protein) and myelin basic proteins, which are major targets in MS autoimmunity, are evident. This evidence concerns the gene MBP and myeloid sarcoma.