Rataj et al. demonstrated that ZFP36L1 was markedly suppressed in breast cancer cells and patient tissues and that a derivative of ZFP36L1 fused to cell-penetrating peptide inhibited the proliferation, migration, invasion, and anchorage-independent growth in vitro and impaired the tumor growth and EMT markers, including Snail, Vimentin, and N-cadherin, in vivo [32]. This evidence concerns the gene ZFP36L1 and breast carcinoma.