Similar to TTP, ZFP36L1 phosphorylation and inactivation by the p38–MK2 axis has been shown to stabilize Nanog and Klf4 in triple-negative breast cancer cells, resulting in breast cancer stem cell phenotype, a feature of chemotherapy-resistance in triple-negative breast cancer [31]. The gene discussed is ZFP36L1; the disease is triple-negative breast carcinoma.