IFNAR1 and Zika virus infectious disease: ZIKV infection at embryonic day 6.5 (E6.5) or E7.5 in either wild-type (WT) immunocompetent mice treated with MAR1-5A3, a blocking anti-Ifnar monoclonal antibody, or in Ifnar1 deficient mice can recapitulate the CZS phenotype in vivo [8].