DPYSL2 and neuroblastoma: The first study indicating that SUMOylation of CRMP2 regulates sodium channel trafficking and biophysical properties showed that expression of a non‐SUMOylatable CRMP2 (CRMP2‐K374A) reduced NaV1.7 current density and surface expression in the catecholamine A differentiated neuroblastoma cell line, and reduced sodium current density in DRG neurons.