Interestingly, in this comparison of LOX-high (n = 31) and LOX-low (n = 28) patients, only a higher proportion of t-AML and MDS-related AML (p = 0.049), higher numbers of monocytic AML according to FAB-subtype (p = 0.003), and more patients with cytogenetically defined high-risk abnormalities (p = 0.001) remained as statistically significant different in these groups in univariate analysis. Here, LOX is linked to acute myeloid leukemia.