Furthermore, we observed that regardless of whether they were treated with P4/Org or not, the viability and proliferative capacity of PC‐9‐mPRα− cells was significantly lower than that of PC‐9 cells (Fig 5), again indicating that knockdown of mPRα expression inhibited the growth of lung adenocarcinoma PC‐9 cells. This evidence concerns the gene PAQR7 and lung adenocarcinoma.