For semaphorin 3C, SEMA3C, a gene overexpressed in glioma and employed by glioma stem cells to promote tumorigenicity [38], we observe 14 NCCMs (in 11 patients; one or more NCCM/patient) with an equal distribution of intergenic and intronic NCCMs (7 each) (Fig. 4a, Additional file 6: Table S5). Here, SEMA3C is linked to glioma.